Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question?
Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question?
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Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question? “Recent evidence shows that SARS-CoV-2 is capable of affecting the genetics and dynamics of erythrocytes and this coexists with a non-homeostatic function of cardiovascular, respiratory and renal systems during COVID-19. In hypothesis, SARS-CoV-2-induced systematical alterations of erythrocytes dynamics would constitute a set point for COVID-19-related multiple organ failure syndrome and death.”
Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question?
Is SARS-nCoV-2-associated systemic…
Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question?
Is SARS-nCoV-2-associated systemic micro-clotting due to spike protein-induced hemolysis resulting in amyloid plaque formation? Can Plasmodium falciparum help us answer this question? “Recent evidence shows that SARS-CoV-2 is capable of affecting the genetics and dynamics of erythrocytes and this coexists with a non-homeostatic function of cardiovascular, respiratory and renal systems during COVID-19. In hypothesis, SARS-CoV-2-induced systematical alterations of erythrocytes dynamics would constitute a set point for COVID-19-related multiple organ failure syndrome and death.”